Shaq’s condition appears to be a simple inflammation, an overuse issue.  He isn’t getting younger after all…  Parker’s injury, a muscle contusion, was clearly a result of a collision.  If it is a significant bruise, it could take a good four to six weeks for him to get back up to speed.  The trouble then is that Parker’s calf muscles will be somewhat de-conditioned, making them vulnerable should he return to play too early.  Of course, with playoffs already in sight, that is exactly what is likely to occur.

Achilles ruptures, on the other hand, are not generally associated with contact.  Most who suffer a ruptured Achilles feel or hear a “pop and think they might have been kicked.  Surgery is often performed quickly, to approximate the two ends of the tendon before the tissue shortens permanently.

What is the Achilles tendon?

Tendons attach muscles to bones.  The Achilles is thicker and more fibrous than most tendons and it connects the calf muscles (the bulky Gastroc in the upper calf and the smaller Soleus closer to the ankle) to the heel bone (calcaneus).  The Achilles tendon and calf muscles are put on stretch when the ankle is flexed up and it is shortened when the toes are pointed.  A tight Achilles or one that is overstretched can predispose to rupture.

If you think you have a tight Achilles, before stretching vigorously, it is important to identify whether the problem is actually with the tendon or if it lies with restricted ankle mobility.  Have a therapist check the range of motion at your ankle with the knee flexed.  The Gastroc muscle crosses the back of your knee as well as the ankle.  With the knee bent, the gastroc muscle is in a shortened position and will not restrict movement at the ankle.  Compare theses findings with the amount of ankle flexion range with the knee straight (with the Gastroc on stretch).  The latter is a measure of flexibility.  If your ankle mobility is significantly restricted, no amount of calf stretching will lengthen your Achilles tendon or even stretch your calf muscles.  The motion simply isn’t accessible to allow that to occur.

What are the consequences of Achilles rupture?

In addition to the pain and swelling that are expected with any sports injury, disruption of the connection between the calf muscles and the heel results in an inability to contract these muscles; That means that you cannot rise up on your toes or walk with a normal push off when weight-bearing on your injured side. Running and jumping are therefore also out of the question.   However, it may be possible to actively point your toes while you are not bearing weight due to the action of other muscles that help to provide this movement.

Why do Achilles tendons rupture?

There is an area of the Achilles that has less blood flow than the rest and it is thought that this section of the tendon may not be as strong.  As we age, tendons, as other tissues of the body, become less supple and may degenerate.  These are some reasons why tendinitis becomes more prevalent and ruptures generally occur in people over 30, especially in middle age.  Younger athletes generally experience a higher tear, well above the ankle, where the muscle joins the tendon (musculo-tendinous junction), though they too can experience a tear closer to the heel.

Some sports are more stressful to the calf muscles and Achilles than others.  Those that require the powerful push-off needed for running and jumping place the greatest demand, as do those like racket sports which entail a lot of stutter-steps and quick stops and starts.  Men are far more likely to suffer Achilles ruptures than women and it is thought that obesity adds to the stress on the Achilles, increasing risk of rupture.  Though this is not a factor for most elite athletes, football linemen and centers built like Shaq do place proportionately greater demand on the tendon.

Other predisposing factors for weekend warriors like you and me include stepping up activity suddenly and significantly  – either by increasing the intensity, frequency or duration of participation or perhaps beginning a new activity without preparation.  All of these increase the stresses placed on the Achilles. It is thought that weakness of the calf muscles, particularly the Soleus, may also be a factor causing the Achilles to rupture; The Soleus can be strengthened by pointing the foot — against resistance — while the knee is bent (as in a sitting position).  A history of multiple steroid injections at the Achilles may be to blame in some cases of rupture.  Be aware that manufacturers of certain medications, such as the antibiotic Levaquin, name tendon issues as a potential side effect.  Inform your physician if you begin to feel Achilles symptoms after beginning a new medication.

One exercise I strongly suggest you avoid is strengthening the Gastroc muscles of the upper calf by hanging the mid-foot and heel off a step or raised platform and doing heel-raises from this position of maximum stretch.  Repeatedly lowering the heel below the level of the step (with your body weight and gravity loading the Achilles tendon), puts the tendon at greater risk of rupture and jeopardizes the structures of an unstable mid-foot.  This is an exercise I see so often in the gym.  Yes, it is important to strengthen the calf, but it is recommended to do so from the more neutral foot–flat position.

How is an Achilles rupture diagnosed?

Physical exam is often pretty conclusive.  The first test is simply to squeeze the belly of the calf muscles and observe whether the foot points as the muscles are manually shortened.  Another obvious sign is when the examiner can move the ankle excessively into a dorsiflexed position (toes toward your nose) – with the knee straight.  As previously discussed, this motion is generally restricted by the tightness of the Achilles and in the case of rupture it is not.  A third test involves observing the motion of a needle inserted into the tendon as the foot is passively moved up and down.  An ultrasound exam and/or positive x-ray findings — particularly those in a lateral (side) view – support the diagnosis.

What are the treatment options?

Small tears may do well with immobilization.  Casting is generally done with the foot in a pointed position, which shortens the tendon allowing it to heal.  Bracing that restricts motion is an alternative. Athletes are not good candidates for conservative management, and those who do not undergo surgery should expect a long recovery (up to a year) before returning to sports.

Operative treatment is the gold standard for athletes, younger patients and those with a complete rupture.  Both treatments are followed by periods of decreased weight-bearing, though the surgical patient progresses at a much faster rate.  Rehab includes work on overall strengthening, cardiovascular conditioning and flexibility, as well as a focus on restoration of normal mobility in the ankle and foot, which become restricted from prolonged immobilization.

Outcomes

According to Wheeless’ Textbook of Orthopaedics, non-operative patients have an average re-rupture rate of 18% and can expect a decrease in strength and muscle endurance of 30%.  83% of surgical patients and 69 % of immobilization patients can expect to resume their pre-injury level of activity.  Wheeless also reported that 93 % of surgical patients were satisfied with the results of treatment, while only 66% of conservatively managed patients felt likewise.  Because of the positioning of the foot with the tendon in a shortened position while casted or braced, non-operative treatment generally does not restore the Achilles tendon to its full length.  In contrast, if immobilized with the ankle in a neutral position, the tendon is generally lengthened, leading to a poor outcome.

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Here’s a key excerpt from the NY Times blog post:

“But a major new review article, published last Friday in The Lancet, should revive and intensify the doubts about cortisone’s efficacy. The review examined the results of nearly four dozen randomized trials, which enrolled thousands of people with tendon injuries, particularly tennis elbow, but also shoulder and Achilles-tendon pain. The reviewers determined that, for most of those who suffered from tennis elbow, cortisone injections did, as promised, bring fast and significant pain relief, compared with doing nothing or following a regimen of physical therapy. The pain relief could last for weeks.

But when the patients were re-examined at 6 and 12 months, the results were substantially different. Over all, people who received cortisone shots had a much lower rate of full recovery than those who did nothing or who underwent physical therapy. They also had a 63 percent higher risk of relapse than people who adopted the time-honored wait-and-see approach. The evidence for cortisone as a treatment for other aching tendons, like sore shoulders and Achilles-tendon pain, was slight and conflicting, the review found. But in terms of tennis elbow, the shots seemed to actually be counterproductive.”

Read the complete article at this link: NTimes.com – Phys Ed: Do Cortisone Shots Actually Make Things Worse?

What is a strain?

A strain results from a pull or overuse of a muscle and entails some level of damage to the fibers of the muscle itself or to the tendon that attaches the muscle to bone.  A Grade I strain, such as the one Pettitte’s MRI confirmed, is the least involved, and implies only mild disruption of the tissue.  In a Grade II injury, some of the fibers of the muscle or musculo-tendinous unit have been torn.  The most severe strain, a Grade III, is actually a complete rupture of the structure.

What are some of the signs and symptoms of a strain?

The first and most noticeable issue, of course, is pain.   The injured area also becomes inflamed, with the signs of inflammation being swelling, redness of the skin and warmth to touch.  Bruising may follow as the internal bleed of the muscle (in grades II and III strains) becomes more superficial.  Not only is there likely to be pain with use of the involved muscle, but pain may be present at rest as well.  A consequence of this swelling and pain is the inhibition of the muscle, resulting in weakness.

What muscles are involved in a groin strain?

The most commonly implicated muscles are the adductors, which is a group of four muscles comprising the inner thigh.  These muscles are the primary movers of the hip into adduction, or toward the midline, thus bringing one thigh in toward the other.  To help you remember that this is the action of the ADDuctors, it might help to know that the ABDuctors are those hip muscles that move the thigh away from midline.  Think ABDuct, to take away (from your other leg) versus ADDuct, to add to…

Note that issues with the abdominal wall, at the attachment of the abdominals to the pubic bone, may also be involved in a groin strain.  The adductor muscles are not the only ones that may be at fault.  I have alluded to this type of injury in a prior post and will dedicate a future column to this fairly common athletic injury, known as a sports hernia.

Andy Pettitte’s issue however, was reportedly one that is exclusive to the adductors.
The four adductor muscles are:

1. Adductor Brevis:  The word “brevis” implies that this muscle is shorter than its counterpart (longus).  In addition to adducting the thigh, this muscle laterally (outwardly) rotates the thigh as it adducts.  Its less significant role is to assists with flexing the hip.

2. Adductor Longus:  This is the longer muscle, and it originates at a different region of the pubic bone than does Brevis.  It functions in much the same way however.  Longus inserts further down the femur than Brevis, with both attaching to a ridge on the back region of the bone.

3. Adductor Magnus:  Magnus, as the root word implies, is the largest or broadest adductor muscle.  It originates both at the pubic bone and the lower portion of the pelvic girdle in front as well as at the sit bone (ischial tuberosity) in back.  It has a broad area of attachment at its distal (far) end, extending as far as the very end of the femur, just above the medial (inner) aspect of the knee joint.

In addition to this muscle acting to adduct the hip/thigh, its front (anterior) fibers assist in flexing the hip while the rear fibers assist in extending the hip.  Like Adductor Brevis and Longus, Adductor Magnus also plays a small role in lateral rotation.

4. Gracilis:  The most unique of the adductor muscles, gracilis is the only one of the four that crosses both the hip and knee joint.  It too originates at the pubic bone but inserts at the tibia (of the lower leg) below the inner aspect of the knee joint.  Two of the three hamstring muscles – the medial (inner) ones – also attach at this same area.

Because of its orientation, when it contracts drawing its lower portion toward the upper, Gracilis serves primarily as a hip adductor muscle.  However, because it inserts below the knee, it also assists the hamstrings in flexing (bending) that joint.  Unlike the other adductor muscles, Gracilis assists in rotating the femur (and tibia) medially (inward).  If the leg is stationary, the Gracilis acts as a hip flexor when it contracts, bringing the upper portion of the muscle toward the lower rather than the other way around.

What does treatment entail?

As for any inflammatory condition, ice and electric stimulation are initially used to minimize swelling and other complications of inflammation.  Certain types of massage are generally performed to promote healing and control the formation of scar tissue.  Gentle, pain-free stretching is also employed.  After the acute period, it is likely that heat followed by ice is utilized.  Controlled and progressive strengthening is instituted in a pain-free fashion and return to activity is promoted gradually.  Toward that end, Pettitte has reportedly begun a throwing program from increasing distances before attempting to pitch.  By doing so, he initially avoids the long stride length, ballistic motion and extreme follow-through that would aggravate his condition and would be required of him to pitch effectively.

If his coach is correct, Kalin Lucas should give David Beckham a call for a Q & A on what to expect next.  Mr. Beckham was headed to his fourth World Cup, a record for an English soccer player, when he left the field in tears on March 14^th after he too suffered an Achilles rupture.  Beckham wasted no time in having surgery, flying to see a specialist in Finland early the next morning for an Achilles repair.

Both Lucas’ and Beckham’s injuries were non-contact, typical of Achilles ruptures.  Lucas claimed not to have felt or heard the “pop”, often associated with Achilles injury, though he reportedly did think he might have been kicked — a sensation that is also classic for a rupture.  Beckham looked behind him when his injury occurred, as though he too may have suspected a kick to the calf.  Both players also likely experienced sharp pain.  Post-game, a teammate said that Beckham “felt the muscle begin to come up”.  This too is common; it is almost like firmly pulling an elastic band until it breaks and seeing the top half retract.  Surgery is often performed quickly, exactly for that reason – to approximate the two ends of the tendon it is often recommended to act before the tissue can shorten permanently.

What is the Achilles tendon?
Tendons attach muscles to bones.  The Achilles is thicker and more fibrous than most tendons and it connects the calf muscles (the gastroc in the upper calf and the soleus closer to the ankle) to the heel bone (calcaneus).  The Achilles tendon and calf muscles are put on stretch when the ankle is flexed up and it is shortened when the toes are pointed.  A tight Achilles or one that is overstretched can predispose to rupture.

What are the consequences of Achilles rupture?
In addition to the pain and swelling that are expected with any sports injury, disruption of the connection between the calf muscles and the heel results in an inability to contract these muscles; That means that you cannot rise up on your toes or walk with a normal push off when weight-bearing on your injured side. Running and jumping are therefore also out of the question.   However, it may be possible to actively point your toes while you are not bearing weight due to the action of other muscles that help to provide this movement.

Why do Achilles tendons rupture?
There is an area of the Achilles that has less blood flow than the rest and it is thought that this section of the tendon may not be as strong.  As we age, tendons, as other tissues of the body, become less supple and may degenerate.  These are some reasons why ruptures generally occur in people over 30 (Beckham is 34), and especially in middle age.  Younger athletes generally experience a higher tear, well above the ankle, where the muscle joins the tendon (musculo-tendinous junction), though they too can experience a tear closer to the heel.

Some sports are more stressful to the calf muscles and Achilles than others.  Those that require the powerful push-off needed for running and jumping place the greatest demand, as do those like racquet sports which entail a lot of stutter-steps and quick stops and starts.  Men are far more likely to suffer Achilles ruptures than women and though it is thought that obesity adds to the stress to the Achilles, increasing risk of rupture, that is clearly not a factor for elite athletes.

Other predisposing factors for weekend warriors like you and me include stepping up activity suddenly and significantly  – either by increasing the intensity, frequency or duration of participation or perhaps beginning a new activity without preparation.  All of these increase the demand placed on the Achilles. It is thought that weakness of the calf muscles, particularly the soleus, may also be a factor causing the Achilles to rupture; The soleus can be strengthened by pointing the foot — against resistance — while the knee is bent (as in a sitting position).  A history of multiple steroid injections at the Achilles may be to blame in some cases of rupture.  Be aware that manufacturers of certain medications, such as the antibiotic Levaquin, name tendon issues as a potential side effect.  Inform your physician if you begin to feel Achilles symptoms after beginning a new medication.

One exercise I strongly suggest you avoid is strengthening the gastroc muscles of the upper calf by hanging the mid-foot and heel off a step or raised platform and doing heel-raises from this position of maximum stretch.  Repeatedly lowering the heel below the level of the step (with your body weight and gravity loading the Achilles tendon), puts the tendon at greater risk of rupture.  This is an exercise I see so often in the gym.  Yes, you should strengthen the calf, but it is recommended to do so from the more neutral foot–flat position or not fully weight-bearing if on stretch.

How is an Achilles rupture diagnosed?
Physical exam is often pretty conclusive.  The first test is simply to squeeze the belly of the calf muscles and observe whether the foot points as the muscles are manually shortened.  Another obvious sign is when the examiner can move the ankle excessively into a dorsiflexed position (toes toward your nose) – with the knee straight.  This motion is generally restricted by the tightness of the Achilles and in the case of rupture it is not.  A third test involves observing the motion of a needle inserted into the tendon as the foot is passively moved up and down.  An ultrasound exam and/or positive x-ray findings — particularly those in a lateral (side) view – support the diagnosis.

What are the treatment options?
Small tears may do well with immobilzation.  Casting is generally done with the foot in a pointed position, which shortens the tendon allowing it to heal.  Bracing that restricts motion is an alternative. Athletes are not good candidates for conservative management, and those who do not undergo surgery should expect a long recovery (up to a year) before returning to sports.

Operative treatment is the gold standard for athletes, younger patients and those with a complete rupture.  Both treatments are followed by periods of decreased weight-bearing, though the surgical patient progresses at a much faster rate.

Outcomes
According to Wheeless’ Textbook of Orthopaedics, non-operative patients have an average re-rupture rate of 18% and can expect a decrease in strength and muscle endurance of 30%.  83% of surgical patients and 69 % of immobilization patients can expect to resume their pre-injury level of activity.  Wheeless also reported that 93 % of surgical patients were satisfied with the results of treatment, while only 66% of conservatively managed patients felt likewise.  Because of the positioning of the foot with the tendon in a shortened position while casted or braced, non-operative treatment generally does not restore the Achilles tendon to its full length.  In contrast, if immobilized with the ankle in a neutral position, the tendon is generally lengthened, leading to a poor outcome.

The Spartans of Michigan State will meet up with Cinderella Northern Iowa, a ninth seed, this Friday.  Tom Izzo is a great guy and a great coach.  His team has stepped up under pressure before, just as they did on Sunday.  Kalin Lucas will either be with them in spirit or cheerleading from he bench.  Maybe they can pull this one out in his honor.

Though first quoted as having said he hoped to play through the injury, Roy subsequently decided on the surgery saying, “…If I’m going to be out there, I want to be contributing.  If we’re fortunate enough to advance in the playoffs, having the surgery now gives me the best opportunity to help our team.”  Don’t count on it sports fans. There are more than the two obvious “ifs” in that statement and the one between the lines speaks to the improbability that Roy, or anyone for that matter, could recover sufficiently in such a short time so as to play NBA caliber ball, especially without risking the health of his knee over the long haul.  NBA.com later quoted a more realistic Kevin Pritchard, GM of the Trail Blazers, as saying “We are looking forward to Brandon making a full recovery and expect him to be ready for the start of the regular season.”

So, what is a meniscus anyway?
A meniscus is a cartilagenous structure that appears in a few joints of the body, most notably at the knee.  There is an inner, or medial meniscus and an outer, or lateral meniscus.  Both lie between the tibia (the large bone of the lower leg), and the femur (thigh bone).

The medial meniscus is ‘C’ shaped and the lateral meniscus has more of an ‘O’ shape, and they span the knee joint from front to back.  The menisci serve several important purposes — A primary role is to enable the surfaces of the bones that form the knee joint (the femur and tibia) to fit together better (particularly as the knee is bent).  This prevents excess movement between the bones thus assisting in stabilizing the knee.  This increased stability diminishes wear and tear of the joint surfaces, minimizing arthritic changes.  Menisci also distribute the forces at the joint and bear a good deal of the load that is transmitted during movement and with the compressive forces of activity.  They are like the bumper cushions and shock absorbers of the knee.

Why is the medial (inner) meniscus hurt more often than the lateral one?
A compressive force coupled with rotation at the knee as it moves from a bent to a straight position is thought to be the most likely cause of meniscal tears. However, some tears may be considered degenerative in that there is no known trauma.  The latter type are generally diagnosed in an over 40 population.

The medial and lateral meniscus are anchored (via ligaments) to the femur and tibia.  Other ligaments also connect the two menisci to each other.  However, there is a degree of mobility of the menisci, which enhances their function and helps to prevent injury.  Some studies have demonstrated that the rear portion (called the posterior horn) of the medial meniscus has the least amount of mobility, and this may contribute to the frequency with which it is injured.

Another reason the medial meniscus is frequently injured is that it is attached to the medial collateral ligament (MCL), which stabilizes the inner compartment of the knee joint (take a look at a recent entry on Da’Sean Butler for more on the MCL).  As a result, injuries to the MCL also frequently cause a tear of the medial meniscus.

What is the difference between a meniscal repair and a meniscectomy?
A meniscectomy is the removal of the entire meniscus.  This procedure is rarely performed these days because we have a better understanding of the important role that the menisci play in protecting the knee.  However, many people undergo a partial meniscectomy (using an arthroscope), in which a portion of the meniscus is shaved off, to eliminate the torn section.  The choice to preserve the meniscus is made whenever possible and the determining factor is the type of tear as well as its size and location. Some stable small tears may not require surgery at all.

To oversimplify, the outer section of each meniscus has a better blood supply than the more interior region.  Tears in this outer, or vascularized portion are those that respond to repair.  Where the blood supply is limited, sufficient healing will not take place so rather than repair it, a portion of the meniscus is removed.

Another factor that is important in determining whether a meniscus is repaired or partially removed is the stability of the joint.  If a knee is unstable, a meniscal repair is likely to fail.  If an athlete has an ACL (anterior cruciate ligament) tear along with a meniscal tear, the ACL must be reconstructed at the same time the meniscal surgery is performed to ensure its success.

Is there a difference in the way the two surgeries are rehabilitated?
In the old days (I’ve been around awhile!) meniscal repairs were rehabilitated in a slower more guarded fashion than partial meniscectomies, limiting a patient’s weight-bearing and initially bracing the knee in an extended position. This resulted in more muscle atrophy from disuse, increased difficulty restoring full mobility and delayed progress to full function. The current school of thought is to accelerate the rehab process, much like that for the partial meniscectomy.  However, this still takes at least 6-8 weeks, and for most weekend-warriors and major league spectators, longer.

So, back to Brandon Roy… There is no point in pushing his limit.  Rehab, even for high-level athletes should not be rushed, and when players return to competition too soon they often sacrifice long-term health for short-term rewards.  The Trail Blazers are smart in playing it safe and putting a priority on having a healthy Brandon Roy around for future seasons.

What is the difference between the AC joint and the shoulder joint?
The AC joint and the shoulder joint are not one and the same, though they are often referred to interchangeably in the sports pages.  Both are a part of the shoulder girdle. The AC joint, or acromio-clavicular joint, is the connection between the outer end of the collarbone (clavicle) and the hood over the shoulder joint called the acromion.  The acromion is an extension of the shoulder blade (scapula).

The shoulder joint (also called the gleno-humeral joint) is the interface between the upper arm bone (humerus) and its socket (the glenoid fossa), which is also an extension of the shoulder blade. Though the shoulder is the more complex joint, and can therefore suffer a more extensive variety of injuries, the AC joint is also important to the overall mechanics of the shoulder girdle.  An unstable (too loose) or restricted (too tight) AC joint can be a precursor to pain, a decline in function, abnormal movement and a number of shoulder pathologies.

FYI: There is one more joint in the shoulder girdle, and that is where the inner end of the clavicle is attached to the breastbone (sternum).  It is called the sternoclavicular (SC) joint.  The SC joint is more protected because of its central position; it is also less mobile and is rarely injured.

 What is an AC sprain?
Ligaments connect one bone to another, providing stability at joints.  In the case of any sprain, a ligament is stretched, sometimes to the point of tearing it completely.  There are two sets of ligaments that stabilize the AC joint – one set securing the clavicle to the acromion (which is next to it and above the shoulder joint) and another securing it to a small bone below called the coracoid process.  The coracoid is yet another (though smaller) extension of the shoulder blade.

Trauma, generally resulting from a fall onto the involved shoulder with the arm at the side, can sprain the clavicular ligaments.  AC sprains come in several varieties – those that tear both sets of ligaments outright are the most dramatic and the clavicle becomes elevated because its anchors are ruptured.  These are called AC separations.  In Grade I AC sprains the ligaments are injured but remain essentially intact, while Grade II sprains result in tears of some but not all of the ligaments.  That is why in a Grade II sprain there is no obvious deformity. Both Grade I and II sprains are treated conservatively, meaning that they do not require surgery.  In Grade III sprains both sets of ligaments are ruptured and the stability of the AC joint is significantly compromised; the greater the instability, the more likely the need for surgery.  AC injuries that are even more severe may result in the clavicle being displaced into the soft tissue behind it.  These are Grade IV sprains (though in some rating systems Grades V and VI describe the most severe injuries). 

Why didn’t Sam Bradford have surgery after his initial injury?
It was reported that Bradford’s opening game AC sprain was a Grade II or III.  He returned to play faster than you or I might have (not that I’ve played football lately), but this course of action was not out of the ordinary for a serious athlete.  Had a hard tackle to his midsection not forced him to land on that shoulder so soon afterward, Bradford would likely have recovered sufficiently to make it through the balance of the season relatively unscathed.  Re-injury evidently exacerbated his condition so much so that surgery was the best option to move forward with confidence.

According to published reports, Bradford was cleared to throw only five weeks before his Pro Day earlier this week.  General Managers and coaches were out in force to see if he could still put on a show, and that is exactly what Bradford did.  Known for his passing accuracy, velocity and high completion to interception ratio, the consensus appeared to be that Bradford hadn’t lost a thing.  The recycled and hard-working Bradford is also said to have added between 12 and 20 pounds of muscle to his upper body (accounts have varied) since his injury and with his rehab. Bill Devaney, GM of the soon-to-pick-first St. Louis Rams, was reportedly very impressed.  The rest of us will have to wait and see if he was impressed enough.

What is fascia?
Fascia is a fibrous connective tissue that occurs throughout the body and overlays the muscles, organs, nerves and blood vessels.  It acts as a restraint to keep our tissues and structures in place. If you have ever prepared chicken you have seen the filmy white tissue between the skin and the meat – that is fascia.  At the bottom of the foot, (the plantar surface), the fascia is particularly fibrous and connects the heel bone to the toes.  It is prone to stresses or small tears that can result in an inflammatory (“itis”) response.

Common causes of Plantar Fasciitis
Fasciitis usually develops over time rather than from trauma and the problem is often stubborn, especially if left untreated. Tightness of the calf muscles and Achilles tendon can predispose to plantar fasciitis, as can tightness of the fascia itself.  A loss of mobility at the ankle or the big toe (not being able to flex up/back sufficiently) can also lead to fasciitis by changing our movement patterns, reducing the shock absorbency of the joints, and limiting the ability of the calf and fascia to stretch to their fullest, resulting in tightness.  Additionally, weakness of the muscles of the calf or foot/ankle impact function and lead to injury by placing more stress on all the related tissues.  Other typical pieces of the cause and effect puzzle may include structural malalignment (such as a high or low arched foot) or the presence of a heel (bone) spur.

Women who wear high heels much of the time are prone to fasciitis because the calf muscles and fascia become tight due to the positioning of the foot and ankle.  People whose work requires that they spend an inordinate amount of time standing or walking (particularly on hard surfaces) and those who are significantly overweight are also more prone to plantar fasciitis.

Symptoms of Plantar Fasciitis
Swelling and tenderness to pressure or touch are generally complaints of those with fasciitis, as is a sharp pain in the heel area or arch of the foot.  One of the most common complaints is morning pain, particularly with the first steps from bed. During a prolonged period of rest, where the foot and ankle are relaxed (with the toes pointed downward), the plantar fascia and muscles of the calf are on slack.  The sudden stretch imposed by putting the foot on the floor and walking can trigger significant pain.  The same might occur after sitting for an extended period.  In very mild cases, after walking a bit and warming up the tissues, pain subsides. Taj Gibson, another Chicago Bull suffering from plantar fasciitis has been receiving treatment but has been able to play through it, an obvious sign that his condition is not as severe as Joakim Noah’s.

Prolonged walking and especially running, which requires a more forceful push-off, are likely to increase the pain and inflammation associated with fasciitis.  Jumping will do likewise.  Not the best scenario for Joakim Noah who, when he began his medical leave, was the NBA’s second leading rebounder.

TREATMENT
Treatment of plantar fasciitis requires a multifaceted approach to reduce inflammation, stimulate healing, stretch tight tissues, strengthen weak musculature and minimize additional stresses.  Here are some tried and true methods of care as well as some of the emerging techniques:

Rest – time off from offending activities is an important part of treatment.

Night Splint – wearing a night boot to keep the ankle at a 90-degree angle maintains some stretch of the calf and fascia and significantly reduces early morning pain.  By helping to lightly stretch tight structures over a prolonged period, resolution of plantar fasciitis is accelerated.

Deep tissue massage – performed manually and also by rolling the foot on massagers or things such as a tennis ball or frozen can of soda can be very helpful.  Deep massage with the ankle held at 90 degrees and the big toe held gently in a backwards-bent position can help to ease fascial pain and tightness.

Stretching – tight calf muscles and fascia must become more flexible to limit recurrence.

Range of motion – treatment to restore motion where it is limited may be necessary to allow for more flexibility of the attached structures.

Cushioned heel lifts – by slightly elevating the heel, stress on the tight structures is minimized during walking or prolonged standing, and the increased shock absorption of a heel lift also decreases the stress from impact.  Choice of footwear factors in here as well (notice that many surgeons and chefs wear clogs).

Ice and electric stimulation – both help to reduce inflammation and swelling

Iontophoreses – another very helpful tool used in physical therapy.  For plantar fasciitis, “ionto” involves the use of a corticosteroid medication that is driven through the skin in the region of the inflammation with a transistor size electric stimulator.  It is painfree, entails only localized use of the drug and does not entail an injection.

Taping – even after returning to competition, taping techniques are used to support the arch.  Taping can be very helpful and enable a player to compete without aggravating a condition as well as reduce the chance for recurrence after a problem resolves.

Strengthening weak muscles throughout the lower body and especially those of the calf and foot is crucial, as is building muscular endurance.  For example weak toe flexors (muscles that let you grip with your toes) are a common problem resulting in fatigue in the foot and strain on the fascia due to poor push-off.  Even the toes must be strengthened when treating foot and ankle conditions.

Balance training is vital as well, working to stabilize while standing on one foot and challenged to reach or bend.  Balance training should be progressed from stable to unstable surfaces to increase the difficulty of the tasks.

Arch supports – if flattened or high arches or other malalignment issues are part of the problem, over-the- counter or custom orthotics (shoe inserts) may be recommended.  It is generally suggested to wait until you have increased your flexibility (if it is one of the causative factors) before getting a custom orthotic because the resting position of the foot may change once greater flexibility is achieved.

Prolotherapy – Prolotherapy treatment involves injecting a sugar water solution into the involved ligament or tendon where it attaches to the bone. This is intended to cause a localized inflammation in these affected areas in order to then increase the blood supply and flow of nutrients.  It is thought to stimulate the tissue to repair itself.  I’m not sold.

Plasma Rich Platelet Therapy (PRP)– Joakim Noah recently underwent PRP and shock wave treatments. Both are relatively new.  Some recent studies on PRP were not as favorable as the original clinical impressions, showing that it was no more effective than injecting saline. Platelets are one of the four components of blood.  The others are red and white blood cells and plasma. The procedure involves taking a patient’s own blood, spinning it down to the platelets, which are said to release proteins called growth factors, then injecting that back into the injured area.  The treatment is thought to accelerate tissue and wound healing.

Shock Wave Therapy – Another emerging treatment, shock waves are said to work by inducing microtrauma to the affected tissue, which then stimulates a healing response. This healing response causes a repair process during which small blood vessels form to increase delivery of nutrients to the affected area.

Injections – Not on my go-to list of treatments for plantar fasciitis, they are still in the arsenal so are included here.  The cortisone injection is occasionally needed to jump start the rehab process by reducing inflammation and pain sufficiently so that the active treatments (such as stretching and strengthening) can be progressed without exacerbating symptoms.  With the potential side effects of injection always at issue, it should not be a first line of defense or used repeatedly in one area of the body.  Nor should cortisone be the entire treatment even if it brings relief.  That is dangerous because it gives a false sense of order when in fact the underlying causes of the problem have yet to be addressed.  Cortisone by injection at the foot is even riskier because of the more fragile nature of the tendons there and their predisposition to rupture.

Surgery – A very last and infrequently relied upon resort, surgery to lengthen the fascia is sometimes the final step when all else has failed.

Wait a minute, isn’t the UCL at the elbow?
Recent columns on this site have discussed UCL injuries to pitchers’ elbows resulting in the need for Tommy John surgery as well as the implications of ankle ligament tears.  First let’s quickly review the basics to help you understand the nature of Shaq’s injury.

A ligament is the connective tissue connecting one bone to another to provide stability at a joint.  Each joint has at least one ligament on each side.  In the hand or elbow, the inner (medial) side, or side of the ulna bone in the forearm, is called the ulnar side and the outer (lateral) side is called the radial side because of its relationship to the radius (the outer bone in the forearm). Hence, at the base of the thumb, just as at the elbow, the ligament stabilizing the innermost part of the joint is called the ulnar collateral (UCL).

Injury to a ligament diminishes stability at the joint it protects, and may do so even on a permanent basis, so Shaq’s prior injury may have predisposed him to re-injury. 

Other names for the UCL tear of the thumb
Most often called “Skiers’ Thumb” these days because of its prevalence on the slopes, this injury used to be known as “Gamekeepers Thumb” because it was commonly found amongst Scottish Gamekeepers (wildlife managers) as a result of a repetitive stress and stretch to the ligament that they suffered when doing their work. 

How does this injury occur?
A force that pulls the thumb away from the hand places stress on the UCL at the joint where the thumb meets the palm.  If the force at this joint (also called the MCP, or metacarpophalangeal joint) is traumatic and causes the joint to exceed its normal limit of movement, the UCL is sprained. And, as for any ligament, a Grade III Sprain is really a complete tear.

A skier who falls while holding the pole, or anyone who tries to break a fall by reaching out with his hand to absorb the impact may suffer a Skiers’ Thumb injury.  If the thumb is bent backwards and a ligament tears, it is unlikely to be able to heal in the anatomical position because of the resultant instability of the joint.  That is why Shaq is having surgery.  Occasionally, just as was previously discussed with regard to ligament injuries at the inner aspect of the ankle, the UCL ligament may not tear but instead might pull off a chip of bone at its point of attachment (at the base of the thumb).  This injury is called an avulsion fracture and it too can result in instability at the joint if not corrected surgically.

So, what did Shaq’s thumb probably feel like after the injury?
Just as after any partial or complete ligament tear, fluid builds up causing swelling.  Visible discoloration is an indication that a ligament (or other vascularized structure like muscle or tendon) was torn.  Naturally, pain is a factor and it will hurt to use the thumb (to grip or squeeze) or to bend it backward.  Reports are that Shaq will be out for six to nine weeks to heal and rehab following today’s surgery.  The Cavs or course, are hoping to have him back in the line-up to help them in the later rounds of the playoffs.

First, what is a sprain?
You may recall from our discussion of the Tommy John procedure that a ligament is connective tissue that connects one bone to another at a joint to provide stability at that joint.  When a ligament is put on stretch it is considered sprained, and these traumatic injuries are labeled as either Grade I, II or III sprains depending on their severity.  A Grade III sprain IS a complete tear of the ligament – they are one and the same thing.

“High” versus “Low” ankle sprains:
The “low ankle sprain” is a relatively new term and one that is not used in the medical literature.  It is thrown about in the sports pages to distinguish the most common type of sprain from the more serious “high ankle sprain”.  The high sprain is a disruption of the connective tissue that attaches the bones of the lower leg (tibia and fibula) to each other in the area above the ankle joint.  As for all sprains, these are also classified as Grade I, II or III.  We will address the high ankle sprain in a later blog.

Type of sprains most commonly seen:

Lateral (Inversion) Sprains:
There are three ligaments supporting the outer ankle – one toward the front, one toward the back and one central ligament.  The outer ankle is sprained more frequently than the inner compartment for many reasons.  This type of sprain is called a lateral or inversion sprain and it occurs when a player rolls the ankle outward.  Because of the position of the foot at the time of injury, most often it is the front and central outer ligaments that are injured.

The ligaments of the outer ankle are not as substantial or strong as those supporting the medial compartment.  When a player lands on another player’s foot while coming down from a rebound (as Kobe did on January 14^th), it is easy to exceed their limit and wind up with a sprain. The nature of the ligaments themselves, coupled with lack of control of the landing position on an unstable surface sets the stage.

Medial (Eversion) Sprains:
The ligament on the inner side of the ankle is a broad, thick structure that is infrequently sprained.  Movement into eversion (rolling in) is limited to begin with and when this area is stressed, the ligament is so strong that it is more likely that its boney attachment will splinter off (this is called an avulsion fracture) than that the ligament will sprain/tear.  Occasionally, as for Anthony Randolph of the injury prone Golden State Warriors, a player will suffer both a lateral sprain (in his case a grade III of two outer ligaments) as well as an avulsion of the inner side.  This, boys and girls, is a very big problem.

Some predisposing factors to consider:
When team orthopedists perform physicals there are many things they examine to determine if an athlete has a history of sprains or an inclination toward having them.

1. A laxity of the ligaments is a dead give-away.  Ligaments don’t rebound like rubber bands – once stretched, they remain loose and therefore the joint they are intended to protect becomes vulnerable.  Scar tissue that forms in the healing process may provide some stability but is not as supple and isn’t a great substitute.
2. Tight tissues or joints. One weak link can cause a rippling effect. For instance, limited mobility of the first joint of the big toe can predispose a player to ankle sprains!  If the toe can’t extend back as it should, a player may have no choice but to roll outward at the ankle and onto the outer border of the foot when landing. 
3. Strength of the foot and ankle musculature (as well as that of the entire lower extremity).  Strength should be normal and similar on the right and left.
4. Single-leg balance.  Balance is evaluated with eyes open and closed as well as with challenges imposed.  If strength or balance is compromised, sprains might result.
5. Agility – the ability to quickly change direction – so important in running and cutting during sport.

Treatment & Healing Time
All of the above factors are also addressed in treatment of a sprain. However, athletes often push themselves to return to competition before they are ready.  Certainly they do so more than the rest of us (if we are smart anyway).  The daily pressure to play, the lure of the big game or being in the final year of a contract can be motivators that we will never know firsthand.  By example, Kobe Bryant aggravated his ankle on February 3^rd when Lamar Odom stepped on his foot as he was pivoting, again causing stress to the ligaments.  All the treatment and tape wasn’t enough to withstand the stress to the area – it simply hadn’t yet healed.

The severity of a sprain clearly impacts the healing time required.  Though even high school athletes often tape up and return to play within days of a Grade I sprain and 3-4 weeks of a Grade II injury, full healing can take a very long time.  In fact, studies show that ligament healing takes at least a year and that is why it is often said that a clean fracture (one that doesn’t require surgery) may be preferable to a Grade III sprain.  In a recent review of 31 studies done by van Rijn, it was reported that 5-33% of Grade III sprainers still had pain one year after injury and that full recovery was documented in only 36-85% of cases within three years (meaning 15-64% did not recover fully in that time).  He reported that 5-33% still experienced pain at one-three years and that 3% – 34% of the patients studied were at risk of re-spraining. Serious business.

Generally, after injury, the emphasis of early treatment is to reduce swelling and inflammation. This progresses to functional treatment rather than immobilization. In most cases for non-professional athletes, return to sports participation occurs when they have full ankle range of motion, at least 90% strength on the injured side and can run and change direction full-out and without pain.